Full name: | Ribosomal RNA small subunit methyltransferase G |
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Synonym: | gidB |
GI: | 121190 |
COG: | COG0357 |
UniProt: | P25813 |
Structures: | | 1XDZ | |
Alpha Fold Predicted Structure: | AF-P25813-F1 |
Enzyme type: | methyltransferase |
Position of modification - modification: |
s:533(527) - m7G |
Title: | |
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Classification: | |
Technique: | |
Pdb Files | 1XDZ.pdb   |
Pdbx/mmCIF Files | 1XDZ.cif   |
MNIEEFTSGLAEKGISLSPRQLEQFELYYDMLVEWNEKINLTSITEKKEVYLKHFYDSITAAFYVDFNQVNTICDVGAGAGFPSLPIKICFPHLHVTIVDSLNKRITFLEKLSEALQLENTTFCHDRAETFGQRKDVRESYDIVTARAVARLSVLSELCLPLVKKNGLFVALKAASAEEELNAGKKAITTLGGELENIHSFKLPIEESDRNIMVIRKIKNTPKKYPRKPGTPNKSPIEG
RsmG is responsible for the synthesis of m7G535 (position 527 in E. coli) in the universally conserved loop of hairpin 18 in domain I. This modified nucleotide plays an essential role in binding antibiotics like streptomycin and kasugamycin. However the dependence on RsmG for antibiotic binding depends on the activity of RsmA (KsgA) catalyzing the formation of m6,6A1518,1519 at the 3'-end of 16S rRNA by RsmA (KsgA). In B. subtilis, mutations in both RsmA and RsmG allow better cell viability in the presence of antibiotics. AdoMet is the methyl group donor.
Reaction | Substrate | SubstrateType | Position | (Anti)Codon | Modified (Anti)Codon | Amino Acid Change | Transcript Name | Transcript Region | Cellular Localization | References |
---|---|---|---|---|---|---|---|---|---|---|
G:m7G | RNA | rRNA | 535 | LSU-16S | Prokaryotic Cytosol | 17573471    |
Alpha Fold Pdb Files | AF-P25813-F1.pdb   |
Alpha Fold Pdbx/mmCIF Files | AF-P25813-F1.cif   |
DSSP Secondary Structures | P25813.dssp   |
Title | Authors | Journal | Details | ||
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Identification of the RsmG methyltransferase target as 16S rRNA nucleotide G527 and characterization of Bacillus subtilis rsmG mutants. | Nishimura K, Johansen SK, Inaoka T, Hosaka T, Tokuyama S, Tahara Y, Okamoto S, Kawamura F, Douthwaite S, Ochi K | J Bacteriol | [details] | 17573471 | - |
Inactivation of KsgA, a 16S rRNA methyltransferase, causes vigorous emergence of mutants with high-level kasugamycin resistance. | Ochi K, Kim JY, Tanaka Y, Wang G, Masuda K, Nanamiya H, Okamoto S, Tokuyama S, Adachi Y, Kawamura F | Antimicrob Agents Chemother | [details] | 19001112 | - |